Author Question: A client with H. pylori asks the nurse why bismuth (Pepto-Bismol) has been prescribed along with ... (Read 67 times)

Anajune7 · **on:** Jun 25, 2018

A client with H. pylori asks the nurse why bismuth (Pepto-Bismol) has been prescribed along with oral antibiotics for treatment. What should the nurse explain about the use of bismuth (Pepto-Bismol) for treatment of this health problem?

Select all that apply.
A) It helps prevent the side effects of antibiotics.
B) It increases stomach acid to help kill bacteria.
C) It helps relieve ulcer-related constipation.
D) It is effective with inhibiting bacterial growth.
E) It keeps bacteria from sticking in your stomach.

Question 2

A nurse is educating a client with peptic ulcer disease (PUD) regarding Helicobacter pylori and its role in PUD. Which statements by the nurse are appropriate to include in the teaching session?

Select all that apply.
A) The bacteria produces enzymes that improves the efficacy of mucous gel in protecting the gastric mucosa.
B) Helicobacter pylori infection is found in about 25 of individuals with PUD.
C) Your inflammatory response to H. pylori contributes to gastric cell damage.
D) H. pylori infection increases production of gastric acids.
E) H. pylori infection is spread by droplets in the air.

nikmaaacs · **Reply #1 on:** Jun 25, 2018

Answer to Question 1

Answer: D, E

Bismuth compounds (Pepto-Bismol) are added to the antibiotic regimen to inhibit bacterial growth. Bismuth compounds prevent H. pylori from adhering to the gastric mucosa. Bismuth does not prevent all side effects of antibiotics. Bismuth does not increase stomach acid. Bismuth is used to relieve diarrhea, not constipation.

Answer to Question 2

Answer: C, D

Helicobacter pylori infection, found in about 70 of individuals who have PUD, is unique in colonizing the stomach. It is spread individual to individual (oral-oral or fecal-oral) and contributes to ulcer formation in several ways. The bacteria produce enzymes that reduce the efficacy of mucous gel in protecting the gastric mucosa. In addition, the host's inflammatory response to H. pylori contributes to gastric epithelial cell damage without producing immunity to the infection. H. pylori infection increases the production of gastric acids.