Answer to Question 1
4
Rationale 1: Atropine occupies muscarinic receptors.
Rationale 2: Atropine blocks parasympathetic actions of acetylcholine.
Rationale 3: Atropine does not potentiate acetylcholine effects on nicotinic receptors.
Rationale 4: Atropine is a cholinergic-blocking agent that occupies muscarinic receptors.
Global Rationale: Atropine is a cholinergic-blocking agent that occupies muscarinic receptors. It is classified as an anticholinergic agent because it inhibits the effects of the parasympathetic nervous system, which induces the fight-or-flight responses of the sympathetic nervous system.
Answer to Question 2
1
Rationale 1: Potassium chloride (KCl) must never be administered intravenous (IV) push, as bolus injections can overload the heart and cause cardiac arrest.
Rationale 2: Potassium chloride must never be administered via intravenous (IV) push, even if slowly, as cardiac arrest may result.
Rationale 3: Although tissue necrosis may occur, this is not the primary concern.
Rationale 4: Renal failure is not the most concerning outcome of administering potassium chloride intravenous (IV) push.
Global Rationale: Potassium chloride (KCl) must never be administered intravenous (IV) push, as bolus injections can overload the heart and cause cardiac arrest. Potassium chloride must never be administered via intravenous (IV) push, even if slowly, as cardiac arrest may result. Cardiac failure, not renal failure, is the most likely outcome of administering potassium chloride intravenous (IV) push. Although tissue necrosis may occur, this is not the primary concern.