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Author Question: The nurse explains that atropine (Atropair) increases heart rate in which manner? 1. Blocking the ... (Read 63 times)

JMatthes

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The nurse explains that atropine (Atropair) increases heart rate in which manner?
 
  1. Blocking the beta receptors of the parasympathetic nervous system.
  2. Directly stimulating the sympathetic nervous system.
  3. Potentiating the effects of acetylcholine on nicotinic receptors.
  4. Blocking the effects of acetylcholine by occupying muscarinic receptors.

Question 2

The physician orders potassium chloride (KCl) intravenous (IV) for the patient. The nurse administers this drug intravenous (IV) push. What will be the most likely outcome for this patient?
 
  1. The patient will most likely experience cardiac arrest.
  2. The patient will not experience adverse effects if the push was given slowly.
  3. The patient will most likely experience tissue necrosis at the injection site.
  4. The patient will most likely experience renal failure.



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mohan

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Answer to Question 1

4
Rationale 1: Atropine occupies muscarinic receptors.
Rationale 2: Atropine blocks parasympathetic actions of acetylcholine.
Rationale 3: Atropine does not potentiate acetylcholine effects on nicotinic receptors.
Rationale 4: Atropine is a cholinergic-blocking agent that occupies muscarinic receptors.
Global Rationale: Atropine is a cholinergic-blocking agent that occupies muscarinic receptors. It is classified as an anticholinergic agent because it inhibits the effects of the parasympathetic nervous system, which induces the fight-or-flight responses of the sympathetic nervous system.

Answer to Question 2

1
Rationale 1: Potassium chloride (KCl) must never be administered intravenous (IV) push, as bolus injections can overload the heart and cause cardiac arrest.
Rationale 2: Potassium chloride must never be administered via intravenous (IV) push, even if slowly, as cardiac arrest may result.
Rationale 3: Although tissue necrosis may occur, this is not the primary concern.
Rationale 4: Renal failure is not the most concerning outcome of administering potassium chloride intravenous (IV) push.
Global Rationale: Potassium chloride (KCl) must never be administered intravenous (IV) push, as bolus injections can overload the heart and cause cardiac arrest. Potassium chloride must never be administered via intravenous (IV) push, even if slowly, as cardiac arrest may result. Cardiac failure, not renal failure, is the most likely outcome of administering potassium chloride intravenous (IV) push. Although tissue necrosis may occur, this is not the primary concern.





 

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