Author Question: A nurse is providing discharge instructions to a client with iron deficiency anemia who is ... (Read 54 times)

809779 · **on:** Jun 25, 2018

A nurse is providing discharge instructions to a client with iron deficiency anemia who is experiencing glossitis. Which statements will the nurse include in the discharge teaching for this client?

Select all that apply.
A) Monitor the condition of the lips and tongue daily.
B) Use an alcohol-based mouthwash every 2-4 hours.
C) Provide frequent oral hygiene.
D) Apply a non-petroleum-based lubricating jelly or ointment to the lips after oral care.
E) Use a soft toothbrush or sponge to provide oral care.

Question 2

A nurse is educating a client with anemia about the pathophysiological mechanisms of anemia. Which should be excluded in the nurse's teaching plan for this client?

A) Altered hemoglobin synthesis.
B) Altered DNA synthesis.
C) Decreased hemolysis.
D) Bone marrow failure.

vish98 · **Reply #1 on:** Jun 25, 2018

Answer to Question 1

Answer: A, C, E

Glossitis, inflammation of the tongue that may cause the tongue and lips to turn red, and cheilosis (fissures or cracks at the corners of the mouth) may occur with nutritional deficiencies of iron, folate, and vitamin B12. Client education should include monitoring the condition of lips and tongue daily and providing frequent oral hygiene with a soft-bristle toothbrush or sponge. The client should not use an alcohol-based mouthwash, as this would worsen the glossitis. The client should use a petroleum-based lubricating jelly or ointment to the lips after oral care.

Answer to Question 2

Answer: C

The pathophysiological mechanisms of anemia include altered hemoglobin synthesis, altered DNA synthesis, bone marrow failure, and increased hemolysis. Altered hemoglobin synthesis is the mechanism involved in iron deficiency anemia, Thalassemia, and chronic inflammation. Altered DNA synthesis is the mechanism involved in Vitamin B12 malabsorption or deficiency, and folic acid malabsorption or deficiency. Bone marrow failure is the mechanism in aplastic anemia, red cell aplasia, myeloproliferative leukemias, and lymphomas.