Question 1
At the molecular level, what are the effects of nicotine on the nervous system? How are these effects exerted? How are these effects similar to or different from those of curare? How do these effects explain some of the physiological consequences of smoking?
Question 2
In a laboratory experiment, adding curare, which binds to acetylcholine receptors, to the solution around a muscle decreases the size of the end-plate potential. Adding prostigmine, an acetylcholinesterase blocker, increases the size of the end-plate potential. Explain why.