Author Question: When educating a group of nursing students on the mechanism of action of various antiarrhythmic ... (Read 44 times)

formula1 · **on:** Jul 23, 2018

When educating a group of nursing students on the mechanism of action of various antiarrhythmic drugs, the nurse identifies which of the following drugs as inhibiting the beta-adrenergic receptors of the heart and kidney?

A) Propafenone
B) Amiodarone
C) Disopyramide
D) Acebutolol

Question 2

A client on antiarrhythmic drug therapy complains of nausea, vomiting, abdominal pain, diarrhea, and a ringing sensation in the ears. Which of the following drugs should the nurse consider as the possible cause?

A) Lidocaine
B) Quinidine
C) Flecainide
D) Procainamide

kbennett34 · **Reply #1 on:** Jul 23, 2018

Answer to Question 1

Ans: D
Feedback:
The nurse should inform the nursing students that acebutolol acts by inhibiting the beta-adrenergic receptors of the heart and kidney. Propafenone and amiodarone appear to act directly on the cardiac cell membrane, and not on the beta-adrenergic receptors of the heart and kidney. Disopyramide acts by decreasing the depolarization of the myocardial fibers, and not the beta-adrenergic receptors of the heart and kidney.

Answer to Question 2

Ans: B
Feedback:
The nurse should consider the drug quinidine as the cause for these adverse reactions. Quinidine toxicity is called cinchonism. Some of its symptoms include ringing in the ears (tinnitus), hearing loss, headache, nausea, vomiting, abdominal pain, dizziness, vertigo, and lightheadedness. Lidocaine, flecainide, and procainamide do not cause tinnitus or hearing loss.